Most people weren't getting the information they needed about SARS-CoV-2 through standard media, publications, or interactions.
As SARS-CoV-2 has spread, we've seen numerous variants emerge, and generally only the most significant ones reach the media. However, there are hundreds of thousands of SARS-CoV-2 variants. You can see these variants in the GSA database, a global database where scientists from around the world submit sequences of new SARS-CoV-2 variants they discover. To access this database, you must request permission. You can also check out Nextstrain, which gives you a glimpse of what's happening in the GSA database.
As SARS-CoV-2 has evolved, it has become increasingly transmissible. For example, a subvariant of BA 286, which is a subvariant of Omicron, is currently being developed along with many others. Due to the large number of variants of this highly contagious respiratory virus, we will likely need continuous vaccine updates to address these new variants.
A common myth is that as viruses mutate, they become less deadly. This may be true for a virus so deadly that it kills the host before it can spread effectively. However, in the case of SARS-CoV-2, it spreads very effectively before the host is hospitalized and can die. Therefore, there is no selective pressure for SARS-CoV-2 to become less deadly. Rather, there is selective pressure for it to become more evasive to the immune system and more transmissible.
Viruses and variants that are more transmissible tend to become the dominant variants. Technically, we're still in a pandemic, although we're no longer in the emergency phase where we feared the infection would overwhelm hospitals and cause chaos. Let me show you what this looks like in a CDC graph. These are weekly deaths in the United States in blue versus test positivity in orange. You can see how they're fairly well matched up until the peak of the Omicron wave, after which we see a divergence: people are still getting sick, but on average we have fewer weekly deaths.
There are several reasons for this. The most vulnerable people, many of them, didn't make it. Many people have been exposed to the virus, which educates the immune system on how to deal with it, whether through infection, vaccination, or both. In addition, we have medications like paxlovid and molnupiravir, which can stop viral replication and have been shown to reduce hospitalization, death, and the development of long COVID.
Although the chart shows improvement, SARS-CoV-2 remains the only respiratory virus among the leading causes of death in the United States each year. In 2022, it was the third leading cause of death, surpassing car accidents. In 2023, it will likely be the fourth leading cause of death. Much of this is preventable if people stay up-to-date on their vaccinations.
SARS-CoV-2 is very different from the flu. Not only is it more deadly, but it infects more types of cells in the body because it uses the ACE2 receptor, which is present on many epithelial cells. All blood vessels in the body have these receptors, so the virus can be present throughout the body, even after a person feels recovered. There has been evidence of persistent virus in brain tissue, the gut, and around muscle fibers, which can have lasting impacts.
There is evidence of higher rates of heart attacks following SARS-CoV-2 infection, even a year or more later. Some preliminary studies suggest a possible link between SARS-CoV-2 and the development of cancer. In addition, the virus can cause dysfunction and dysregulation of the immune system, leaving people somewhat immunocompromised for 12 to 18 months after infection. It can also cause mitochondrial dysfunction, which is linked to accelerated aging and extreme fatigue.
It is estimated that approximately one in ten people suffer some form of long COVID. Although some people recover, many experience a variety of long-term symptoms. Aside from the physical damage to organs, which can accumulate over time, these persistent effects underscore the severity and complexity of SARS-CoV-2 infection.
• Remember: covid is not over, 50% of infections are asymptomatic, minimum 10% of infections end up in long COVID, re-infections wreck us, COVID spreads and moves like cigarette smoke, think of the people around you and you as people who are all day smoking, it becomes more visual to understand how COVID moves.
• There is no way to “train” the immune system because it is not a muscle. there is a common misconception that exposure to harmful germs strengthens the immune system. viral diseases like COVID, flu, measles weaken the immune system, leaving the possibility of lasting damage. The reality is that you don't build your immunity with repeated infections, vaccines strengthen the immune system by teaching it to recognize pathogens without all the risks. Focusing on infection prevention is key.
• Rapid antigen tests give many false negatives.
• Solving the pandemic was never in the cards for the capitalist world.
• Instead, the explicit goal of the ruling class has been to make the pandemic simply disappear from public perception. Any reminder of the existence of a highly-transmissible, highly-dangerous, mass-disabling disease could trigger panic, or worse: organized, militant labor action. Averting this crisis required a careful campaign of culture-crafting; the people themselves needed to become convinced that there was no reason to fight. Consent for protracted mass infection needed to be manufactured.
“The cold truth of the matter is that the motive behind COVID minimization is greed and social control. (…) Solving the pandemic was never in the cards for the capitalist world. Instead, the explicit goal of the ruling class has been to make the pandemic simply disappear from public perception.” Let Them Eat Plague! http://clarion.unity-struggle-unity.org/
#MaskUp #WearAMask #CovidRealist #CovidIsAirbone #LongCovid #YallMasking #DisabledLiberation #DisabilityJustice
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