REVOLUTION @ Exeter, NH 🌟 “Creation Myths”

Drawing common purpose @ a BIG WATCH event / April 2026

https://marekbennett.com/2026/04/20/revolution-exeter/

What sunlight bouncing off of cars has to do with happiness. For some people. Marketing expert #BryanHaut on how we experience happiness. #YoureOnMuteThePodcast Episode 8: https://podcasts.apple.com/us/podcast/episode-08-bryan-haut/id1746606151?i=1000655744407 #memory #happiness #podcast
What sunlight bouncing off of cars has to do with happiness. For some people. Marketing expert #BryanHaut on how we experience happiness. #YoureOnMuteThePodcast Episode 8: https://podcasts.apple.com/us/podcast/episode-08-bryan-haut/id1746606151?i=1000655744407 #memory #happiness #podcast

Vaibhav (VB) Srivastav (@reach_vb)

ChatGPT와 Codex의 메모리 기능이 사용자 취향과 맥락을 반영해 출력 품질을 높인다고 평가했다. 특히 Codex가 어떤 테스트를 돌리고 커밋 흐름을 따를지 세세한 작업 습관까지 이해하는 점을 언급해, AI 코딩 에이전트에서 장기 메모리와 개인화 컨텍스트가 실무 가치를 크게 높인다는 인사이트를 제공한다.

https://x.com/reach_vb/status/2055879083737677837

#chatgpt #codex #memory #agent #coding

Vaibhav (VB) Srivastav (@reach_vb) on X

Memories in both ChatGPT and Codex are godsend! It allows them to contextualise and hyper specialise outputs and generations to you and your “taste” I’m often amazed by how Codex would just know tiny details of which tests to run, what commit dynamics it should follow and how to

X (formerly Twitter)

Soveryn Intelligence (@Soveryn_AI)

로컬 LLM 배포를 위한 메모리 시스템을 만들고 있다고 밝힌 트윗입니다. 로컬 에이전트/LLM 앱에서 장기 대화 맥락과 상태 관리를 해결하려는 초기 단계의 개발 언급으로 볼 수 있습니다.

https://x.com/Soveryn_AI/status/2056037043864559698

#llm #memory #localdeployment #agent

Soveryn Intelligence (@Soveryn_AI) on X

@daniel_mac8 literally building a memory system that works for local LLM deployment its the first step

X (formerly Twitter)

Forgetfulness

I did not post on Friday. Forgetfulness? I suppose, combined with Life imposing other things onto my daily calendar.

It happens. In his poem, “Forgetfulness,” Billy Collins is focused on how memory weakens with age. I can identify.

My habit of rereading novels that I enjoyed in my youth and finding them to be new material is said by Collins in this way:

The name of the author is the first to go
followed obediently by the title, the plot,
the heartbreaking conclusion, the entire novel
which suddenly becomes one you have never read,
never even heard of,

It’s “normal” when you get to a certain age to be more forgetful. (What age? It depends on the individual.) I accept that, but I try very hard to concentrate and bring back that forgotten thing. “Don’t tell me,” I tell my wife when I can’t recall a name or word. I want to force my brain to make that connection again. Sometimes it happens. Sometimes.

As Collins says, “Whatever it is you are struggling to remember / it is not poised on the tip of your tongue.” Where is it? Like the lost car keys, it is somewhere, and I feel that I should be able to find it. The poet is not as hopeful.

It has floated away down a dark mythological river
whose name begins with an L
as far as you can recall,

And Billy ends with my greatest fear about my memory and my aging brain:

well on your own way to oblivion where you will join those
who have even forgotten how to swim and how to ride a bicycle.

https://youtu.be/n-a8ELOVig4?si=BVFkfirb-Sr9HWCd

#BillyCollins #forgetfulness #memory
Forgetfulness

The name of the author is the first to go

Poets.org

Experts no longer believe in photographic memory. Here’s what convinced them

https://www.sciencefocus.com/the-human-body/photographic-memory-wrong

A Science Focus longread article by me on the science of eidetic (photographic) memory, and how the brain deals with memory in general.

#Brains #Memory #Neuroscience

Experts no longer believe in photographic memory. Here’s what convinced them | BBC Science Focus Magazine

Though there are people capable of incredible feats of memory, true photographic memory has never been proven to exist

BBC Science Focus Magazine

Boosting Mitochondrial Activity Restores Memory in Mouse Models of Dementia

📰 Original title: Scientists reversed memory loss by recharging the brain’s tiny engines

🤖 IA: It's clickbait ⚠️
👥 Users: It's clickbait ⚠️

View full AI summary: https://en.killbait.com/boosting-mitochondrial-activity-restores-memory-in-mouse-models-of-dementia.html?utm_source=mastodon_world&utm_medium=social&utm_campaign=killbait.mastodon_world

#neuroscience #mitochondria #dementia #memory

Boosting Mitochondrial Activity Restores Memory in Mouse Models of Dementia

Researchers from INSERM and the University of Bordeaux, in collaboration with the Université de Moncton, have demonstrated for the first time that mitochondrial dysfunction may directly contribute to cognitive decline in neurodegenerative diseases. Using a novel tool called mitoDREADD-Gs, designed to temporarily stimulate mitochondrial activity within neurons, scientists were able to restore memory performance in mouse models of dementia. This finding suggests that energy deficits in neurons may occur before brain cells die, highlighting a potential early intervention target for conditions like Alzheimer's disease. Mitochondria, the energy-producing structures of cells, are critical for neurons to communicate and maintain memory function. When their activity drops, neurons lose efficiency, which may underlie memory and cognitive deficits. While the research is still in early stages and limited to animal models, it points to a new therapeutic avenue focusing on restoring cellular energy rather than solely targeting classic pathological markers such as amyloid plaques or tau tangles. Future research aims to assess whether prolonged mitochondrial stimulation can prevent neuronal loss, slow disease progression, or delay symptom onset in humans. The study emphasizes the importance of neuronal energy health in brain function and offers a fresh perspective for developing treatments for neurodegenerative diseases.

KillBait

Boosting Mitochondrial Activity Restores Memory in Mouse Models of Dementia

📰 Original title: Scientists reversed memory loss by recharging the brain’s tiny engines

🤖 IA: It's clickbait ⚠️
👥 Users: It's clickbait ⚠️

View full AI summary: https://en.killbait.com/boosting-mitochondrial-activity-restores-memory-in-mouse-models-of-dementia.html?utm_source=mastodon_social&utm_medium=social&utm_campaign=killbait.mastodon_social

#neuroscience #mitochondria #dementia #memory

Boosting Mitochondrial Activity Restores Memory in Mouse Models of Dementia

Researchers from INSERM and the University of Bordeaux, in collaboration with the Université de Moncton, have demonstrated for the first time that mitochondrial dysfunction may directly contribute to cognitive decline in neurodegenerative diseases. Using a novel tool called mitoDREADD-Gs, designed to temporarily stimulate mitochondrial activity within neurons, scientists were able to restore memory performance in mouse models of dementia. This finding suggests that energy deficits in neurons may occur before brain cells die, highlighting a potential early intervention target for conditions like Alzheimer's disease. Mitochondria, the energy-producing structures of cells, are critical for neurons to communicate and maintain memory function. When their activity drops, neurons lose efficiency, which may underlie memory and cognitive deficits. While the research is still in early stages and limited to animal models, it points to a new therapeutic avenue focusing on restoring cellular energy rather than solely targeting classic pathological markers such as amyloid plaques or tau tangles. Future research aims to assess whether prolonged mitochondrial stimulation can prevent neuronal loss, slow disease progression, or delay symptom onset in humans. The study emphasizes the importance of neuronal energy health in brain function and offers a fresh perspective for developing treatments for neurodegenerative diseases.

KillBait