Show threadLorraineMay 4

@rbreich
anybody who couldn’t see this was the part of the plan from the beginning, doesn’t understand these people very well

haven’t even read Project 2025 & knew

Jonathan Taplin’s book goes a bit into it, but also looking back at 16th century France & the North Korean regime will give a good sense of what they envision for us

manufacturing consent has captured us all

#neoliberalism is how we got here & this is the result of Biden’s handling of SARSCov2 not based on science as promised #CovidPapers but from the advice of a marketing company

you can blame Biden’s advisors , primarily Jeff Zeinst, for paving the way for fascism as it’s prob not a coincidence it happened last after the 1918 Spanish Flu

blame Biden

Show threadLorraineApr 3

https://zeroes.ca/@Brad/114271977539130851
Brad’s got all the #CovidPapers 😍🥰🙏🏼

idk why everyone wants so badly to remain in denial when you have some agency over your future

this thing where 1 person is doing the entire group project is not sustainable

get with the program people🖤

we are begging you🌈

Brad Mitchell (@Brad@zeroes.ca)

April 2, 2025- “COVID-19 may put patients at risk for other infections for at least 1 year”- https://www.cidrap.umn.edu/covid-19/covid-19-may-put-patients-risk-other-infections-least-1-year

zeroes.ca
Show threadLorraineMar 25

Whole-body visualization of SARS-CoV-2 biodistribution in vivo by immunoPET imaging in non-human primates | Nature Communications

https://www.nature.com/articles/s41467-025-58173-y

March 21, 2025

it’s like an album drop… #covidPapers

Whole-body visualization of SARS-CoV-2 biodistribution in vivo by immunoPET imaging in non-human primates - Nature Communications

There are limited approaches to monitor virus spread in vivo. Here, the authors report PET/CT-based in vivo imaging to track SARS-CoV-2 biodistribution in a COVID-19 non-human primate model using a radiolabeled human antibody revealing persistent detection in the lung and brain 3 months after infection.

Nature
LorraineDec 10, 2024

Equivocating and Deliberating on the Probability of COVID-19 Infection Serving as a Risk Factor for Lung Cancer and Common Molecular Pathways Serving as a Link

https://www.mdpi.com/2076-0817/13/12/1070

my new edited🧵listing all the important papers that have come out over the past 4 years

actually understood >90% of this thanks to a couple books & lectures prior to reading

every abstract starts the same.. in 2019 SARSCov2 first appeared…
you do not want cancer in any form, yet so many pathways via Covid #ace2 #CovidPapers published December 6, 2024

Equivocating and Deliberating on the Probability of COVID-19 Infection Serving as a Risk Factor for Lung Cancer and Common Molecular Pathways Serving as a Link

The COVID-19 infection caused by SARS-CoV-2 in late 2019 posed unprecedented global health challenges of massive proportions. The persistent effects of COVID-19 have become a subject of significant concern amongst the medical and scientific community. This article aims to explore the probability of a link between the COVID-19 infection and the risk of lung cancer development. First, this article reports that SARS-CoV-2 induces severe inflammatory response and cellular stress, potentially leading to tumorigenesis through common pathways between SARS-CoV-2 infection and cancer. These pathways include the JAK/STAT3 pathway which is activated after the initiation of cytokine storm following SARS-CoV-2 infection. This pathway is involved in cellular proliferation, differentiation, and immune homeostasis. The JAK/STAT3 pathway is also hyperactivated in lung cancer which serves as a link thereof. It predisposes patients to lung cancer through myriad molecular mechanisms such as DNA damage, genomic instability, and cell cycle dysregulation. Another probable pathway to tumorigenesis is based on the possibility of an oncogenic nature of SARS-CoV-2 through hijacking the p53 protein, leading to cell oxidative stress and interfering with the DNA repair mechanisms. Finally, this article highlights the overexpression of the SLC22A18 gene in lung cancer. This gene can be overexpressed by the ZEB1 transcription factor, which was found to be highly expressed during COVID-19 infection.

MDPI
Show threadLorraineJul 1, 2024

I really do not understand people.
Their complete lack of empathy for others & imagination

Did they not read enough as children? Are they delusional enough to think they are health superior & nothing will ever happen to them?

Getting Covid over & over has become a rite of passage.. something to commiserate over, to bond over🙄 then to laugh off

It was a giant mistake not to link #sarscov2 #viralPersistence #reinfection with #HIV

Complex stuff bores people so a clickbait statement is required

I will patiently wait for the #science #covidPapers to come out, & keeping in mind I’ve never even taken basic biology, but after observing this disease in myself & my spouse then peering out at an endless supply of people constantly exposing themselves…

have concluded that “clearing” remnants of virus to very low levels is key to long term survival

Not clearing it accelerates aging & all the fun diseases that come w/old age

We aren’t seeing it in large numbers in <30 years old, but that’s because they started w/a newer “car” & their endothelium hasn’t been wrecked as much by lifestyle

So it’s going to pick off the older & more vulnerable, then work its way down the age bracket

I know I’m right, but will patiently wait for the studies or the results I suppose

But all of this could be prevented by #wearADamnMask

LorraineApr 22, 2024

@douglaspynn @qurlyjoe
Interesting comparison

I guess when your body is breaking down, regardless of impetus, it all breaks down the same way
Irony of all this.. we live in a culture obsessed w/youth & not aging yet…

My go-to #CovidPapers was the telomere one from what seems an eternity ago, but nope🙄

Watching The Hunger at 15 really did a number on me because although I’m ambivalent myself about aging, David Bowie being tucked away into the attic is quite the nightmare

Show threadLorraineApr 21, 2024

While on the subject of horror movies… watched this video of Dr. Greger’s book signing from a couple months ago promoting his new book “How not to Age” a follow-up to “How not to Die” which I’m currently slogging through eye-rolling the entire way while listening to it🙄🙄🙄🙄🙄

Can’t decide which title is more ironic tbh.. #longevity

I quite liked him, but for someone who uses science studies to back up his advice, it seems he is completely ignoring the hundreds of #CovidPapers

Anybody whose brand is built on health that🚫😷… I cannot comprehend it & surreal doesn’t even begin to describe it

https://www.youtube.com/watch?v=qCbTakHuj3M

@douglaspynn

Show threadLorraineApr 11, 2024

@brianvastag Furthermore, altho Covid presents as a cold or allergies if at all for most, there are too many #CovidPapers that show the innumerable amounts of harm it does to your vital organs such as brain, heart, kidneys, eyes.. the entire endothelium & vascular system

It’s an accelerated aging disease

We’ve known most of this since 2021-22
This disease has not fundamentally changed

Your might want to recommend #Novavax to your friend, but they prob already know about it

Also… your friend I’m sure doesn’t WANT to live the way they do. I do not love going out looking like this, but in order to not get sick again & because the entire world has decided a neuroinvasive virus “is just a cold”, I willingly get mocked, laughed & stared at like I’m a circus clown. If people understood what is being deliberately hidden from them, I honestly believe they would be more cautious

Until that day happens… I’m really grateful for my #microclimateAir2

I’m the furthest thing from a scientist, but I can read & I treasure my health more than any derision🖤🖤

LorraineApr 1, 2024

I prided myself for decades for being “open-minded” but have decided it’s actually quite dangerous.. in many circumstances

Prob decades ahead of my time to be in my “judge-y bitch era” but no time like the present🖤

If you’re not wearing a damn mask, my judgement is two-fold; not smart & devoid of empathy

Certain things do not require compromise #wearADamnRespirator #aprilFool
#nyc #WeAreStillInAPandemic #CovidPapers

Show threadLorraineMar 13, 2024

@maggiejk
https://heart.bmj.com/content/early/2024/01/24/heartjnl-2023-323483
while we are on the topic of #pulmonaryEmbolism a new #CovidPapers on the subject..

round and round we go🙄
@ABScientist

The role of COVID-19 vaccines in preventing post-COVID-19 thromboembolic and cardiovascular complications

Objective To study the association between COVID-19 vaccination and the risk of post-COVID-19 cardiac and thromboembolic complications. Methods We conducted a staggered cohort study based on national vaccination campaigns using electronic health records from the UK, Spain and Estonia. Vaccine rollout was grouped into four stages with predefined enrolment periods. Each stage included all individuals eligible for vaccination, with no previous SARS-CoV-2 infection or COVID-19 vaccine at the start date. Vaccination status was used as a time-varying exposure. Outcomes included heart failure (HF), venous thromboembolism (VTE) and arterial thrombosis/thromboembolism (ATE) recorded in four time windows after SARS-CoV-2 infection: 0–30, 31–90, 91–180 and 181–365 days. Propensity score overlap weighting and empirical calibration were used to minimise observed and unobserved confounding, respectively. Fine-Gray models estimated subdistribution hazard ratios (sHR). Random effect meta-analyses were conducted across staggered cohorts and databases. Results The study included 10.17 million vaccinated and 10.39 million unvaccinated people. Vaccination was associated with reduced risks of acute (30-day) and post-acute COVID-19 VTE, ATE and HF: for example, meta-analytic sHR of 0.22 (95% CI 0.17 to 0.29), 0.53 (0.44 to 0.63) and 0.45 (0.38 to 0.53), respectively, for 0–30 days after SARS-CoV-2 infection, while in the 91–180 days sHR were 0.53 (0.40 to 0.70), 0.72 (0.58 to 0.88) and 0.61 (0.51 to 0.73), respectively. Conclusions COVID-19 vaccination reduced the risk of post-COVID-19 cardiac and thromboembolic outcomes. These effects were more pronounced for acute COVID-19 outcomes, consistent with known reductions in disease severity following breakthrough versus unvaccinated SARS-CoV-2 infection. Data may be obtained from a third party and are not publicly available. CPRD: CPRD data were obtained under the CPRD multi-study license held by the University of Oxford after Research Data Governance (RDG) approval. Direct data sharing is not allowed. SIDIAP: In accordance with current European and national law, the data used in this study is only available for the researchers participating in this study. Thus, we are not allowed to distribute or make publicly available the data to other parties. However, researchers from public institutions can request data from SIDIAP if they comply with certain requirements. Further information is available online (<https://www.sidiap.org/index.php/menu-solicitudesen/application-proccedure>) or by contacting SIDIAP (sidiap@idiapjgol.org). CORIVA: CORIVA data were obtained under the approval of Research Ethics Committee of the University of Tartu and the patient level data sharing is not allowed. All analyses in this study were conducted in a federated manner, where analytical code and aggregated (anonymised) results were shared, but no patient-level data was transferred across the collaborating institutions.

Heart